Endothelial lesions result in an inflammatory response, with the performance of various cell types (lymphocytes, monocytes, platelets, and smooth muscle cells), [12] causing endothelial cell dysfunction, vascular wall stiffening, and atherosclerosis plaque formation.[13] These endothelial lesions and complications are confirmed by inflammatory markers that result in endothelial activation, such as C-reactive protein (CRP) and interleukin 6 (IL-6), [14,15] this last 1 correlating positively with systolic and diastolic pressures. Here, IL6 is linked to atherosclerosis.