The results showed that GlcN not only inhibited NLRP3 inflammasome but also reduced IL-1β secretion in AIM2-, non-canonical- and NLRC4-inflammasome activated J774A.1 macrophages, which are stimulated by poly(dA:dT) and LPS transfection or by Salmonella infection, respectively (Fig. 2F). The gene discussed is NLRC4; the disease is Salmonella Infections.