It is known that cPLA2 and PLB activity promotes formation of arachidonic acid from cellular membranes, which in turn activates cellular cyclooxygenase and lipoxygenase enzymes to secrete inflammation mediators such leukotrienes, thromboxanes, and prostaglandins, and cellular downstream pathways by PKC activation to control infections [17,37]. This evidence concerns the gene PRRT2 and infection.