Increased cell motility has been observed in colon and breast cancer cells following a decrease in ZNF143 expression.20, 24 Another study demonstrated that overexpression of ZNF143 in gastric cancer cell lines enhanced metastatic potential.43 Strong expression of ZNF143 in lung adenocarcinomas was shown to predict shorter disease‐specific survival with increased Ki‐67 labelling.21 Gonzalez and colleagues showed that ZNF143 as an important regulator for CCAAT/enhancer‐binding protein alpha (C/EBPα) in myeloid cells,23 whose dysregulation is closely regulated to myeloid leukaemia. This evidence concerns the gene MKI67 and lung adenocarcinoma.