We observed that lack of γδT cells not only exacerbates liver fibrosis, but also resulted in elevated serum ALT levels, accompanied with more significant intraheptic leukocyte accumulation, compared to CCl4-treated WT mice (Supporting Figure 2C), suggesting that missing γδT cells might promote the infiltration of inflammatory cells to liver tissue and then lead to the death of hepatocytes. The gene discussed is GPT; the disease is Hepatic fibrosis.