Patients with FOXG1 mutations (duplications/deletions, nonsense, frameshift and missense mutations) have clinical features characterized by postnatal growth deficiency and microcephaly, developmental delay with absent speech, defective social reciprocity, poor sleep, stereotypes, dyskinesia, and severe early onset epilepsy (Brunetti‐Pierri et al., 2011; Guerrini et al., 2012; Yeung et al., 2009). This evidence concerns the gene FOXG1 and epilepsy.