In cancer-associated myofibroblasts, treatment with dexamethasone successfully induces nuclear translocation of GR, resulting in an anti-inflammatory phenotype marked by the repression of IL-1β, monocyte chemoattractant protein 1, C-C motif ligand 5, tumor necrosis factor-α (TNFα) and intercellular adhesion molecules [56]. The gene discussed is NR3C1; the disease is cancer.