Fibroblasts treated, with 75 μM RSV for 48 h exhibited markedly enhanced FAO rates in cell lines with mild FAO deficiency, i.e., the myopathic form of CPT2 or VLCAD deficiency, leading to restore normal flux, while no effect was found in fibroblasts from patients with the severe form of the deficiencies [100]. This evidence concerns the gene CPT2 and very long chain acyl-CoA dehydrogenase deficiency.