TRIM44 was reported to be upregulated in quiescent MM cells in the BM in comparison with reactivated cells.63 Furthermore, CXCL12 expression is elevated in hypoxic tissue through HIF‐1, leading to the recruitment of CXCR4‐positive progenitor cells64; therefore, extravasation of CXCR4‐positve DTCs into the bone marrow may be supported by a hypoxic milieu. Here, TRIM44 is linked to Miyoshi myopathy.