IL-17 not only regulates the oxidative induction, apoptosis, autophagy, and other biological effects by mediating the MAPK, GSK3β, NF-κB, and other related signaling pathways but also plays role synergistically with other related cellular factors, and accelerate the development of pulmonary fibrosis [29]. Here, NFKB1 is linked to pulmonary fibrosis.