On the basis of various AD pathogenesis, a series of therapeutic drugs were developed, including cholinesterase inhibitors, cholinergic receptor agonists, anti-Aβ therapeutic drugs, calcium ion (Ca2+) inhibitors, antioxidant drugs, and anti-free radical drugs, but the effect of treatment was not encouraging especially in brain damage caused by oxidative stress [6, 20]. The gene discussed is BCHE; the disease is Alzheimer disease.