SALL4 and nasopharyngeal carcinoma: Cellular responses to DNA damage, including cell cycle arrest and apoptosis, are important determinants of cancer outcomes following radiation therapy.46 Here, we found that inhibition of SALL4 contributed to sensitize NPC cells to radiation, along with enhanced apoptotic cells, DNA damage, and G2/M arrest, indicating that SALL4 regulates the radiosensitivity by impairing radiation‐induced DNA damage and cellular apoptosis.