Adenosine diphosphate (ADP), a specific platelet activator, triggers degranulation of eosinophils through P2Y12R.17 Our recent study suggested an inhibitory effect of clopidogrel (Clo), an antiplatelet drug that targets the P2Y12R, during eosinophilic inflammation and degranulation.18 Furthermore, LTE4, a P2Y12R and CysLTR1 agonist, induces eosinophilic inflammation following a platelet‐dependent mechanism.19 The gene discussed is P2RY12; the disease is inflammation.