YAP1 and McLeod neuroacanthocytosis syndrome: Consistent with this hypothesis, RNAi‐mediated depletion of YAP1 or pharmacologic inhibition of the YAP1‐TEAD transcriptional complex with verteporfin (Liu‐Chittenden et al, 2012) suppressed cell viability and YAP1 target gene expression in MLS cells.