Consistent with these findings, Chen and colleagues [44] have reported that FOXK2 overexpression in lung cancer-derived cell lines leads to upregulation of E-cadherin and α-catenin along with downregulation of N-cadherin and vimentin (Figure 5), suggesting that FOXK2 might modulate the expression of epithelial and mesenchymal markers closely linked to the EMT process. Here, FOXK2 is linked to lung carcinoma.