The critical involvement of the nicotinic acetylcholine receptor α7nAChR has been shown in the anti‐inflammatory effect of the VN in sepsis, a model of systemic inflammation, and in a model of intestinal inflammation, that is postoperative ileus.7 To investigate the role of α7nAChR in the anti‐inflammatory effect of VNS in food allergy, VNS or sham stimulation was performed in OVA‐sensitized α7nAChR−/− mice as earlier described, prior to the first challenge with OVA (Figure 4A). The gene discussed is CHRNA7; the disease is inflammatory response.