The deleterious effects of testosterone after MI may be due to induction of hypertrophy via androgen receptors [29] and / or increasing apoptosis after MI, since a study found that in patients with heart failure post-MI, the rate of myocyte necrosis and apoptosis was higher in men than in women, which was associated with earlier onset of the disease in males [32]. The gene discussed is AR; the disease is heart failure.