In line with the studies demonstrating that HCMV infection induces glucose uptake [13,14,15,16], HCMV infection was shown to dramatically increase the expression of the adipose tissue-specific glucose transporter type 4 (GLUT4) [16,19], and eliminate the expression of the ubiquitously distributed GLUT1 in infected fibroblasts [16]. The gene discussed is SLC2A4; the disease is cytomegalovirus infection.