A number of studies have demonstrated that TGFβ signaling promotes glioma angiogenesis via the upregulation and activation of various angiogenic factors including VEGF.21, 22 In our study, adding anti‐TGFβ antibody did not result in an increased antiangiogenic effect when compared with DC101 treatment alone, thus confirming that the proangiogenic function of TGFβ is likely to be predominantly mediated via VEGF. Here, VEGFA is linked to glioma.