They confirmed that UBR2 could be internalized into p53 wild-type mBMMSC (p53+/+ mBMMSC) and murine foregastric carcinoma (MFC) cells and increased the expression of UBR2 in these cells, which promoted gastric cancer growth and metastasis by activating wnt/β-catenin pathway. This evidence concerns the gene UBR2 and gastric cancer.