Furthermore, in a series of 115 patients with IFN-γR1, IFN-γR2, and STAT1-deficiencies, respectively, only one single patient developed clinical toxoplasmosis (143) and among 141 patients with IL-12Rβ1deficiency no single patient developed toxoplasmosis (144). This evidence concerns the gene IFNGR2 and toxoplasmosis.