Our current observation that primary AML can be eliminated in an in vivo model by TEG001, without affecting the hematopoietic compartment, is in line with our previous observation that an alteration in the RhoB-CD277J axis, the putative ligand of γ9δ2TCR, is selectively observed in the leukemic but not healthy hematopoietic stem cell [12]. The gene discussed is RHOB; the disease is acute myeloid leukemia.