TSPAN1 and idiopathic pulmonary fibrosis: Early reports proved EMT makes a potential contribution to collagen‐producing fibroblasts and myofibroblasts in IPF, and AECs can transdifferentiate into myofibroblasts when they acquire the mesenchymal phenotype via EMT.29, 30 In this study, we investigate whether the TSPAN1 mediate the transformation of AECs—a new possible mechanism for the pathogenetic of IPF, AECs transformed to fibroblast‐like cell, via the process of EMT in IPF.