CYSLTR1 and infection: The overexpression of these genes suggests a response related to infection, as well as activation of an innate immune response (c3ar, cysltr1, stat3), that could lead to cell death mediated by overexpression of fasl. This canonical pathway is also linked to CD3+ IEL increase and adaptive immune responses mediated by MHC class II genes and immunoglobulin production, which are characteristic of CeD36.