In the recent decade, LINC00599 has been suggested to be involved in physiological and pathological processes, such as hippocampal axogenesis [15], retinal cone survival [15], atherosclerosis [16], neurodevelopmental disorder [8], retinal reactive gliosis [6], myeloid-derived suppressor cell differentiation [18], and cigarette smoking [10]. The gene discussed is MIR124-1HG; the disease is atherosclerosis.