As KLRG1 expression increases as T cells differentiate in response to antigen stimulation [17], and a variety of cancer therapies are predicted to increase T cell differentiation, either directly (e.g., immunotherapy) or indirectly (e.g., immune activation after tumor cell death in chemotherapy and radiation therapy), we searched for all publicly available gene expression data from paired pre- and post-treatment human tumor samples, identifying 21 datasets (Supplementary Table 2). This evidence concerns the gene KLRG1 and cancer.