As shown in Figure 5B, the expression of arg1, cd163, cd206, fizz1, and ym1 genes all correlated with an anti-inflammatory, pro-tumor phenotype, were up-regulated in CD11b+ cells in the ipsilateral hemisphere of glioma bearing mice and were significant reduced upon LPS/IFNγ-MV treatment (with the exception of fizz1). The gene discussed is IFNG; the disease is glioma.