NUP98 molecular rearrangements have been detected in acute myeloid leukemia (AML M1, M2, M4, M5, M7, and therapy‐related AML) and to a lesser extent in myelodysplastic syndromes (MDS, therapy‐related MDS) such as blast crisis‐chronic myeloid leukemia (BC‐CML), chronic myelomonocytic leukemia, juvenile myelomonocytic leukemia, and T‐cell acute lymphocytic leukemia.1, 2, 3 At least 72 NUP98 fusion partner genes have been reported so far in the literature (http://atlasgeneticsoncology.org/Genes/GC_NUP98.html). This evidence concerns the gene NUP98 and chronic myelogenous leukemia, BCR-ABL1 positive.