POC5 and androgen insensitivity syndrome: Improved interaction of POC5A429V with ciliary proteins resulted in the loss of POC5 co-localization with acetylated-α-tubulin, impaired cell cycle progression and cilium retraction These findings open new avenues for the understanding the role of POC5 in AIS at the molecular and cellular levels and suggest that centrosomes and cilia may underly AIS pathogenesis