Despite the absence of data on endothelial function, such as PAI-1, von Willebrand factor, or a disintegrin-like metalloproteinase with thrombospondin type 1 motifs 13 (ADAMTS 13) in our study, endothelial dysfunction might be induced by HSCTs among the AKD patients. The gene discussed is VWF; the disease is endothelial dysfunction.