Remarkably, ALCAT1 protein expression was significantly upregulated by overexpression of the human α‐synuclein protein in the Thy1‐αSyn transgenic mice, implicating a role for ALCAT1 in the etiology of mouse models of PD (Figure 3d, quantified in Supporting Information Figure S2o,p). Here, LCLAT1 is linked to Parkinson disease.