In patients with ST‐segment elevation, myocardial infarction (STEMI) in platelet proteomes, a different expression of proteins was observed that were involved in actin cytoskeleton signalling, integrin signalling and integrin‐linked kinase signalling and activation by the collagen receptor GPVI.36 However, as a control group both studies’ authors used stable chronic ischaemic cardiopathy patients, who also had increased blood platelet activity and may have had changes in proteomes compared to healthy individuals. The gene discussed is GP6; the disease is myocardial infarction.