The protein levels of adiponectin in the EAT were positively correlated with the mitochondrial OXPHOS capacity with complex I and fatty acid-linked substrates (Fig. 3e) and with complex I + II and fatty acid-linked substrates (Fig. 3f), or the maximal ETS capacity with non-fatty acid and fatty acid substrates (Fig. 3g) even in CAD patients. This evidence concerns the gene ADIPOQ and coronary artery disorder.