Osteopetrosis is a group of inherited disorders in which the bones become harder and denser resulting from the dysfunction of osteoclasts.1 The most common and adult‐onset form with benign nature refers to the autosomal dominant osteopetrosis type II (ADOII), also known as Albers‐Schenberg disease.2 The molecular mechanism of ADOII is known as mutations in the voltage‐gated chloride channel 7 (ClC‐7) gene in chromosome 16p13.1 The gene discussed is CLCN7; the disease is autosomal dominant osteopetrosis 2.