We found that Bcl-2 expression in RA was significantly higher than that in the control in pPDGFRαβ+CDH11–, pPDGFRαβ-CDH11+, and pPDGFRαβ+CDH11+ populations (P = 0.0037, P < 00001, and P = 0.0012, respectively; Supplemental Figure 3). The gene discussed is BCL2; the disease is rheumatoid arthritis.