Our analyses showed a 186% increase of the polymeric immunoglobulin receptor (PIGR), a 341% increase of fructose‐bisphosphate aldolase B (ALDOB), and a 22% increase of vitronectin (VTN) in NAFLD patients with NGT (NAFLD‐cohort 1) compared with healthy controls (P < 0.001, Fig 3A and B). The gene discussed is PIGR; the disease is metabolic dysfunction-associated steatotic liver disease.