In a subsequent GWAS, although none of our findings showed a genome-wide significance with SAH in patients with IA, we discovered that there was a suggestive association with the SGCZ gene, which is a component of the vascular smooth muscle sarcoglycan complex that contributes to muscular dystrophy pathogenesis [47] and should be further investigated for its contribution to SAH formation at the molecular level. Here, SGCZ is linked to muscular dystrophy.