Due to the sympathetic system activation in the postprandial period [17], the characteristic postprandial glycemic profile in patients with HA1A subtype would be an accentuation of the postprandial hyperglycemia, followed by a relative decrease in the glycemic value. The first effect would be due to the potentiation of postprandial glucagon-induced hyperglycemia, while the second would be caused by the increase in glucagon effect on insulin secretion and is dependent on insulin reserve [18-19]. Here, GCG is linked to Hyperglycemia.