In nasopharyngeal carcinoma, VTN was identified as once of BPIFB1-interacting proteins and could be reduced by BPIFB1, leading to less formation of VTN-integrin αV complex, suppressing the EMT process, and inhibition of the activation of downstream FAK/Src/ERK signaling pathway [21]. Here, PTK2 is linked to nasopharyngeal carcinoma.