Genotype‒phenotype correlation studies in FA or FA-like patients further demonstrated functional roles for other HR factors including BRCA1 (FANCS), PALB2 (FANCN), RAD51 (FANCR), and FANCJ in ICL repair pathway [51,75,76,77,78,79,80,81,82,83]. This evidence concerns the gene RAD51 and Friedreich ataxia.