GO analyzes revealed that these lncRNAs were involved in inflammatory-related biological processes (regulation of p38MAPK cascade, interleukin-1 beta production, and acute inflammatory response to antigenic stimulus) and neuron-related biological processes (negative regulation of axon extension, negative regulation of neuron projection development, and negative regulation of neuron maturation), which are important in learning and memory [41], as well as the development of AD. This evidence concerns the gene IL1B and Alzheimer disease.