Clezardin et al [41] reported that IPP accumulation in ZA-treated breast cancer cells might be recognized by Vγ9Vδ2 T-cells as tumor phosphoantigens and induce Vγ9Vδ2 T-cell expansion, promote Vγ9Vδ2 T-cell chemotaxis to the tumor, and increase Vγ9Vδ2 T-cell cytotoxicity. The gene discussed is IPP; the disease is neoplasm.