The antioxidative effects of BCL6 in VSMCs were supported by previous findings that BCL6 overexpression inhibited oxidative stress response to etoposide and other chemotherapeutic reagents in B-cell lymphoma cells [18] and that BCL6 knockdown augmented the hypoxia-induced oxidative stress in cardiomyocytes [36]. Here, BCL6 is linked to B-cell non-Hodgkin lymphoma.