Emerging evidence indicated that certain lncRNAs can served as a competitive endogenous RNA (“ceRNA”) to regulate downstream gene expression and biological function.40 For instance, RNCR3 acts as a ceRNA, and form a feedback loop with Kruppel‐like factor 2 and miR‐185‐5p to prevent atherosclerosis.41 TGFB2‐OT1 regulates autophagy in vascular endothelial cells (VECs) via sponging miR‐3960, miR‐4488, miR‐4459.42 Further, our search of miRanda revealed that miR‐224‐5p and miR‐497‐5p can bind to LINC00094 via the putative microRNA response elements (MREs). Here, BRD3OS is linked to atherosclerosis.