CAV3 and persistent truncus arteriosus: To determine whether the preservation of t‐tubular ICa in Cav‐3 OE myocytes helps to maintain Ca2+ release, we investigated the latency and heterogeneity of Ca2+ release along a single t‐tubule from the time of membrane depolarization (Figure 5a), which showed that Cav‐3 OE had no significant effect on latency, nor did it affect the increase in latency of both the initial and maximum rate of rise of Ca2+ observed following TAC (Figure 5b, top), suggesting that TAC‐induced impairment of local Ca2+ release is unaffected by Cav‐3 OE.