The distribution of truncating variants in genes previously associated with dilated cardiomyopathy (e.g. the gene for the sarcomere protein titin) is similar for women with PPCM and persons with idiopathic dilated cardiomyopathy [14], suggesting that in women already predisposed to dilated cardiomyopathy, the stress of pregnancy may simply hasten the process, resulting in PPCM. The gene discussed is TTN; the disease is dilated cardiomyopathy.