However, we acknowledge that, although in the context of sepsis, the suppression of inflammatory response mediated by F. hepatica antigens could be advantageous; this phenomenon carries serious implications for the control of other concurrent infections with F. hepatica. Studies have demonstrated that this parasite suppresses IFNγ altering the response to tuberculosis10,37 and delaying bacterial clearance in bystander infection with Bordetella pertussis35,36. The gene discussed is IFNG; the disease is infection.