Our qPCR and immunoblot data would argue against this assumption, but without further investigation, we cannot be wholly certain; (ii) It should be mentioned that the two recent proteomic analyses (outlined above) documented no alterations in PP2A, this absence of PP2A change may be due to insensitivities in these assays [10,66]; (iii) A study from Zheng et al. [54] revealed that selenomethionine results in increases in PI3K/AKT/GSK3β and neurogenesis in hippocampi of AD mice. This evidence concerns the gene PTPA and Alzheimer disease.