ETV2 and myocardial infarction: In the present study, while we found a significant upregulation of key endothelial genes from CFs upon ETV2 overexpression, we did not detect noticeable morphological changes of ETV2-infected CFs into ECs or EC-like cells, suggesting that direct conversion of non-ECs to ECs by ETV2 is not a major pathway promoting coronary revascularization in the MI heart.