Scratch assay/wound‐healing assay with A549 cells in the presence of Cdh23 EC1–2 (10 μg·day−1) also demonstrated an increased rate of migration via a faster rate of gap closure than the BSA‐treated (10 μg·day−1) A549 cells (Fig. 5D), indicating the significant role of the excretory isoforms in facilitating cell migration in cancer. Here, CDH23 is linked to cancer.